Preliminary experiments reveal conolidine may possibly inhibit specific ion channels, decreasing neuronal excitability and restricting pain alerts. This mechanism is especially pertinent in neuropathic pain, where abnormal signaling triggers persistent soreness. In addition, conolidine appears to affect G protein-coupled receptor (GPCR) pathways integral to discomfort notion. Computat... https://herberth890nan7.wikiannouncement.com/user