Preliminary experiments suggest conolidine might inhibit specific ion channels, reducing neuronal excitability and restricting ache signals. This system is especially pertinent in neuropathic pain, where abnormal signaling brings about persistent discomfort. Moreover, conolidine appears to influence G protein-coupled receptor (GPCR) pathways integral to ache perception. Computational ... https://kemalr233viv0.glifeblog.com/profile